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Correction: Identification of deleterious variants in patients with male infertility due to idiopathic non-obstructive azoospermia

The Original Article was published on 02 April 2022

Correction: Reprod Biol Endocrinol 20, 63 (2022)

https://doi.org/10.1186/s12958-022-00936-z

Following the publication of the original article [1], it was noted that due to a typesetting error the figure images for Figures 1-5 in the PDF version were not updated and an error was found in Table 1.

The correct Figs. 1, 2, 3, 4, 5 and Table 1 are shown below.

Fig. 1
figure 1

Variants of FANCA in NOA16 and NOA50. A The families affected by the variants in FANCA. The red dotted lines indicate mutated positions in the Sanger sequencing results. B Testicular histopathology of NOA16. C The mutated positions of FANCA are conserved among species (red arrows). And the dotted lines indicate the positions of the FANCA variant in the FANCA protein. M, mutation; WT, wild type

Fig. 2
figure 2

The variant of SYCE1 in NOA51. A The family affected by the variant in SYCE1. The red dotted line indicates the mutated position in the Sanger sequencing. B Testicular histopathology. C The mutated position of SYCE1 is conserved among species (red arrows). And the dotted line indicates the position of the SYCE1 variant in SYCE1 protein. M, mutation; WT, wild type

Fig. 3
figure 3

Variants of DMRT1 in NOA22 and NOA25. A The families affected by the variants in DMRT1. The red arrows indicate mutated positions in the Sanger sequencing results. B Testicular histopathology. C The mutated positions of DMRT1 are conserved among species (red arrows). And the dotted line indicates the position of DMRT1 variants in DMRT1 protein. M, mutation; WT, wild type

Fig. 4
figure 4

The variant of PLK4 in NOA42. A The family affected by the variant in PLK4. The red arrow indicates the mutated position in the Sanger sequencing results. B Testicular histopathology. C The mutated position of PLK4 is conserved among species (red arrows). And the dotted line indicates the position of PLK4 variant in PLK4 protein. S_TKC, Serine/Threonine protein kinases, catalytic domain; M, mutation; WT, wild type

Fig. 5
figure 5

Variants of TEX11 in NOA39 and NOA49. A The families affected by the variants in TEX11. The red dotted line indicates mutated positions in the Sanger sequencing results. B Testicular histopathology. C The mutated positions of TEX11 are conserved among species (red arrows). And the dotted lines indicate the positions of TEX11 variants in TEX11 protein. M, mutation; WT, wild type

Table 1 Deleterious variants detected in patients with non-obstructive azoospermia and related clinical phenotypes.

The original article [1] has been corrected.

Reference

  1. Tang D, Li K, Geng H, et al. Identification of deleterious variants in patients with male infertility due to idiopathic non-obstructive azoospermia. Reprod Biol Endocrinol. 2022;20:63. https://doi.org/10.1186/s12958-022-00936-z.

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Correspondence to Hui Jiang, Xiansheng Zhang, Xiaojin He or Yunxia Cao.

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Tang, D., Li, K., Geng, H. et al. Correction: Identification of deleterious variants in patients with male infertility due to idiopathic non-obstructive azoospermia. Reprod Biol Endocrinol 20, 79 (2022). https://doi.org/10.1186/s12958-022-00950-1

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  • DOI: https://doi.org/10.1186/s12958-022-00950-1