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Fig. 3 | Reproductive Biology and Endocrinology

Fig. 3

From: H3K4me3 mediates uterine leiomyoma pathogenesis via neuronal processes, synapsis components, proliferation, and Wnt/β-catenin and TGF-β pathways

Fig. 3

Functional enrichment of H3K4me3-mediated differential expressed genes with altered status in UL vs MM. The most significant (A) biological processes and (B) cellular components were obtained following functional enrichment analysis of common DEGs associated with H3K4me3 instabilities in UL vs MM tissues. C Functional implication of selected hypertrimethylated/upregulated oncogenes (blue squares) and hypotrimethylated downregulated genes (yellow squares). The genes that were not been previously related to UL are in bold

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Reproductive Biology and Endocrinology

ISSN: 1477-7827

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