Fig. 7From: TOP2A deficit-induced abnormal decidualization leads to recurrent implantation failure via the NF-κB signaling pathwayModel for involvement of TOP2A-NF-κB pathway in decidualization. This schematic illustrates our hypothesis that abnormal expression of TOP2A in endometrial stromal cells affect the processes of decidualization and embryo implantation through the NF-κB pathway and result in RIFBack to article page